1. 目的：研究辣椒素对大鼠面部皮肤一氧化氮合酶阳性神经的影响一氧化氮，探讨CAP治疗三叉神经的镇痛机制。

Objective:To research the effect of capsicin on the NOS in rat facial skin, and to explore the analgesic mechanism of CAP in treating prosopalgia thereby.

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2. 目的 研究电针对抑郁症模型大鼠阴茎组织一氧化氮和环磷酸鸟苷的影响。

Objective To investigate the effect of electroacupuncture on nitric oxide and cyclic guanosine monophosphate levels in penile tissues of rats with depression.

3. 目的 观察麦芽醇对大鼠局灶性脑缺血再灌注损伤后诱导型一氧化氮合酶的影响。

ABSTRACT:Objective To investigate the effects of maltol on the expression of iNOS after focal cerebral ischemia reperfusion injury in rats.

4. 探讨神经型一氧化氮合酶抑制剂7-硝基吲唑(7-NI)在烟雾吸入性肺损伤中的作用。40只SD雄性大鼠被随机分为正常对照组(n=8)、烟雾吸入性肺损伤模型组(n=16)和7-NI治疗组(n=16)，建立烟雾吸入性肺损伤模型。7-NI治疗组在致伤后立即腹腔注射7-NI 20 mg/kg(溶于2 ml花生油中)；正常对照组及模型组腹腔注射2 ml花生油。

M{rX*o0 OBJECTIVE: To investigate the effects of neuronal nitric oxide synthase inhibitor 7-nitroindazole (7-NI) on smoke inhalation pulmonary injury in rats. METHODS: Forty healthy male SD rats were randomly divided into three groups: control group (n=8), smoke inhalation model group (n=16) and 7-NI treatment group (n=16). After reproducing the smoke inhalation model, to the 7-NI treatment group rats 7-NI (20 mg/kg in 2 ml arachis oil) was administered by intraperitoneal injection, while in the control group and the model group, 2 ml arachis oil was administered by intraperitoneal injection.

5. 探讨神经型一氧化氮合酶抑制剂7-硝基吲唑(7-NI)在烟雾吸入性肺损伤中的作用。40只SD雄性大鼠被随机分为正常对照组(n=8)、烟雾吸入性肺损伤模型组(n=16)和7-NI治疗组(n=16)一氧化氮，建立烟雾吸入性肺损伤模型。7-NI治疗组在致伤后立即腹腔注射7-NI 20 mg/kg(溶于2 ml花生油中)；正常对照组及模型组腹腔注射2 ml花生油。

After reproducing the smoke inhalation model, to the 7-NI treatment group rats 7-NI (20 mg/kg in 2 ml arachis oil) was administered by intraperitoneal injection, while in the control group and the model group, 2 ml arachis oil was administered by intraperitoneal injection.

6. 目的 观察急性应激对大鼠血小板一氧化氮释放的影响及其机制。

Objective To investigate the effect of water-immersion restraint stress on nitric oxide production and its mechanism in rat platelets.

7. 目的：探讨一氧化氮及内皮素在支气管哮喘发病中的作用。

Objective:To investigate the role of serum nitric oxide and endothelin in bronchial asthmatic attack.

8. 一氧化氮

8. 这些现象被认为与血红蛋白和一氧化氮之间的氧化还原与和结合过程相关。

These phenomena are proposed to be related with the electron transfer and binding process between hemoglobin and NO.

9. 一氧化氮

9. 黄芪对鲤头肾中巨噬细胞的增殖和一氧化氮产量的影响：离体研究。

Effect of Astragalus radix on proliferation and nitric oxide production of head kidney macrophages in Cyprinus carpio:an in vitro study.

10. 因此 ，本文旨在观察茶色素对内皮细胞一氧化氮合酶生成的影响。1 材料与方

The results suggest that the effect of tea pigments on eNOS expression is one of the mechanisms of its antiatheroselerosis.

11. 灵芝孢子；腹根切断：运动神经元；神经营养素-3；一氧化氮合酶；神经元存活；免疫组织化学；原位杂交；大鼠

Ganoderma spore; Ventral root cut; Motor neuron; Neurotrophin_3（NT_3）; Nitric oxide synthase; Neuronal survival; Immunohistochemistry; In situ hybridization; Rat

12. 一氧化氮

12. 目的：探讨一氧化氮的前体物质精氨酸对急性胰腺炎大鼠止凝血指标的影响。

Objective:To evaluate the effect of the arginine on thehematologic disorders of rat acute pancreatitis.

13. 呼出的一氧化氮和PEF变化无显着关系。

No significant relationship was evidenced between exhaled NO and PEF variations.

14. 一氧化氮的近义词

14. 结果：急性胰腺炎期间会损害肺的氧合能力、代谢情况，以及引起乳酸酸中毒和呼出气一氧化氮水平升高一氧化氮，而这些情况在假手术组中显示可被 TEA 所改善。

RESULTS: Deteriorated oxygenation, metabolic and lactate acidosis, as well as exhaled nitric oxide levels occurring during acutepancreatitis, were reduced by TEA to levels observed in sham-operatedanimals.

15. 本实验通过建立重症急性胰腺炎大鼠动物模型，动态观察血浆和肺组织内一氧化氮(Nitric oxide，NO)产生的变化，及诱导型一氧化氮合酶的表达，探讨一氧化氮与重症急性胰腺炎肺损伤的关系，观察生长抑素类似物奥曲肽对肺损伤的治疗作用。

To investigate the possible beneficial effects andthe mechanism of octreotide on the lung injury severe acutepancreatitis rats. The experimental rat model of severe acutepancreatitis was induced by retrograde cholangiopancreatic ductinjection of 5% sodium taurocholate.

16. 一氧化氮

16. 4一氧化氮(Nitric Oxide，NO)供体——钒酸钠(Sodium Metavanadate，vanadate)和硝普钠(Sodium Nitroprusside，SNP)都能够抑制DFO诱导的白血病细胞凋亡，但对CoCl2诱导的细胞凋亡没有抑制作用。

In no alteration of HIF-1αprotein, nitric oxide donors, metavanadate and sodium nitroprusside, significantly abrogated DFO-induced apoptosis.

17.

17. 综合本研究的结果，不只显示了PTPs 半胱胺酸亚硝基化作用重要的基本分子机制，并且证实了一氧化氮藉由亚硝基化作用影响PTPs活性而扮演重要细胞生理调控的角色。

Our results not only reveal the fundamental basis for the mechanistic detail of Cys nitrosylation of PTPs, but offer insights into a novel biological role of NO that may govern tyrosine phosphorylation-dependent signaling through regulation of cellular PTPs.

18. 所有纳入病例均排除以下患有引起继发性骨质疏松症的各种内分泌疾病者（如柯兴氏病、甲状腺功能亢进、甲状旁腺功能亢进、甲状腺囊肿或机能低下、糖尿病等）；其他严重疾病干扰骨代谢者；测试前3个月内未使用过雌激素、降钙素、一氧化氮、糖皮质激素等干扰骨代谢或对内皮素有影响作用的药品；测试前1年内无骨折；患有严重心脑血管等与内皮素水平相关密切之疾病及肝肾功能异常者。

All patients with secondary osteoporosis caused by all endocrine diseases including Cushing's disease, hyperthyrosis, hyperparathyroidism, capsula glandulae thyroideae or thyroid hypofunction and diabetes, and patients whose bone metabolism was interfered by grave diseases were excluded. Three months before the trial, the patients did not receive any drug that could interfere the bone metabolism or affect endothelin, such as estrogenic hormone, calcitionin, nitrogen monoxidum, glucocorticoid. In addition, the patients suffering from bone fracture within 1 year or grave cardio-cerebrovascular disease, which had intimate correlation with endothelin, liver and kidney disfunction were eliminated.

19. 放疗副作用自由基损伤会影响相矢基因的产物表达，如：一氧化氮合成必需酶NOS，抗凋亡基因h－2，抑癌基因P53 ％D肿瘤血管生成密切相矢的VEGF氧气报警器，抗氧化剂则有一定的抑制作用。

We also find that the local radiation can cause the more expression of some genes such as p53 and NOS, at the same time reduce the expression of bcl-2 and VEGF. But the antioxidants have the reverse effect.

20. 目前仅有少量文献报道一氧化氮（nitric oxide甲烷报警器，NO）在肾脏血管反应性的调控中起一定作用［23］，过氧亚硝基阴离子（peroxynitrite，ONOO-）对肾血管反应性的影响尚未见报道。

Methods IPK was used to examine the effects of renal IR on renal vascular response, the effects of iNOS inhibition on the renal vascular response changes caused by renal IR, and the effects of administration of ONOO- on above parameters.

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